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| Further Reading |
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Bucheli ME,
He X,
Kaplan CD,
Moore CL and
Buratowski S
(2007)
Polyadenylation site choice in yeast is affected by competition between Npl3 and polyadenylation factor CFI.
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Danckwardt S,
Kaufmann I,
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Splicing factors stimulate polyadenylation via USEs at non-canonical 3¢ end formation signals.
EMBO Journal
26:
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Graham RR,
Kyogoku C,
Sigurdsson S et al.
(2007)
Three functional variants of IFN regulatory factor 5 (IRF5) define risk and protective haplotypes for human lupus.
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Hall-Pogar T,
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Specific trans-acting proteins interact with auxiliary RNA polyadenylation elements in the COX-2 3¢-UTR.
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13:
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Kaneko S and
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The mammalian RNA polymerase II C-terminal domain interacts with RNA to suppress transcription-coupled 3¢ end formation.
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Kim Guisbert KS,
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Alternative 3¢ pre-mRNA processing in Saccharomyces cerevisiae is modulated by Nab4/Hrp1 in vivo.
PLoS Biology
5:
e6.
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Kubo T,
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Knock-down of 25 kDa subunit of cleavage factor Im in Hela cells alters alternative polyadenylation within 3¢-UTRs.
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Kyburz A,
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Langen H and
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Direct interactions between subunits of CPSF and the U2 snRNP contribute to the coupling of pre-mRNA 3¢ end processing and splicing.
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23:
195205.
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Millevoi S,
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(2006)
An interaction between U2AF 65 and CF I(m) links the splicing and 3¢ end processing machineries.
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25:
48544864.
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Peterson ML,
Bingham GL and
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Multiple features contribute to the use of the immunoglobulin M secretion-specific poly(A) signal but are not required for developmental regulation.
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26:
67626771.
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Sheets MD,
Ogg SC and
Wickens MP
(1990)
Point mutations in AAUAAA and the poly (A) addition site: effects on the accuracy and efficiency of cleavage and polyadenylation in vitro.
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